IMPETIGO AMPOLLOSO EN NIOS PDF

El impétigo, una de las afecciones de la piel más comunes entre los niños. El impétigo no ampolloso comienza como pequeñas ampollas que se revientan y. Impétigo ampolloso Niños pequeños Siempre causado por S. aureus Por acción de una toxina epidermolítica Ampollas superficiales de. ABSTRACT. Impetigo is a common cutaneous infection that is especially prevalent in children. Historically, impetigo is caused by either group A β- hemolytic.

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The spectrum of the selected antibiotic must cover staphylococci and streptococci, both for bullous impetigo as well as for crusted impetigo. Crusted impetigo located on the arm. Impetigo is a common cutaneous infection that is especially prevalent in children.

Impetigo – review

Systemic absorption is minimal and the little that is absorbed is rapidly converted to inactive metabolite, hence the reason why there are not oral or parenteral formulations available. Unique approaches for the topical treatment and prevention of cutaneous infections: Scalded skin syndrome usually begins after a localized infection on the conjunctiva, nose, navel or perioral region and more rarely after pneumonia, endocarditis and arthritis.

In the United States there is already a formulation of mupirocin ointment without polyethylene glycol. Anti-Bacterial agents; Impetigo; Staphylococcus aureus ; Streptococcus pyogenes.

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El impétigo

It occurs in adults and children but rarely in those under two years of age. Bacterial resistance and impetigo treatment trends: Various streptococci may be commensals on the skin, mucous membranes, and gastrointestinal tract.

The amoxicillin associated with clavulanic acid is the combination of one penicillin with a beta-lactamase inhibiting agent clavulanic acidthus enabling adequate coverage for streptococci and staphylococci. Normal skin is colonized by large numbers of bacteria that live as commensals in its surface or in hair follicles. It is not active against bacteria of the normal cutaneous flora and therefore does not alter the skin’s natural defense.

A crucial factor to the infection virulence is the ability of these bacteria to produce circulating toxins that act as superantigens. Thus, benzathine penicillin or those sensitive to penicillinases are not indicated in the treatment of impetigo.

Dosage of antistreptolysin O may not be useful for cutaneous infections since its titles do not increase satisfactorily.

Its actions against most Gram-positive bacteria are limited. Currently, the most frequently isolated pathogen is S.

In the impetigo treatment review performed by the Cochrane Database of Systematic Reviews, the authors report a relative lack of data on the efficacy of topical antiseptics. Staphylococci are transmitted primarily by hand, particularly in hospital settings. Bacitracin A is the main component of commercial products and is generally formulated as a zinc salt.

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. It is particularly important in the neonatal period, starting usually after the second week of life, although it can be present at birth in case of premature membranes rupture.

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It is the result of Streptomyces fradiae fermentation. Effect of handwashing on child health: Treatment of impetigo does not reduce the risk of glomerulonephritis, but it reduces the dissemination of nephritogenic strains in the population.

Bullous impetigo in diaper area. Fusidic acid in dermatology. It can eradicate S.

Host factors seem to determine the onset of disease. Bacterial skin colonization and infections in patients with atopic dermatitis.

Mupirocin acts by inhibiting bacterial protein synthesis, by binding with isoleucyl-tRNA synthetase enzyme, thus preventing the incorporation of isoleucine into protein chains. Group A streptococci can be subdivided into several serotypes, according to their M protein antigenicity.

These enzymes hydrolyze the beta lactam ring, and they are, so far, the main mechanism of resistance to betalactam antibiotics.